By C. M. Van Der Loos
Academical clinical Centre, Amsterdam, The Netherlands. Describes double and triple immunoenzyme staining tools which are played with commercially to be had reagents. presents regular protocols which are tailored to varied purposes. for brand spanking new researchers within the fields of mobilephone biology, pathology, and histology. define. Softcover.
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88b) ● Demonstration of portosystemic collaterals Fig. 84 Continuous flow reversal in the portal vein (hepatofugal flow) in a patient with liver cirrhosis. Fig. 85 Images of different flow profiles: A, continuous hepatopetal flow; B, pulsatile hepatopetal flow; C, pulsatile flow with brief pulse-synchronous flow reversal (arrows). a In the portal vein. b In the splenic vein. Fig. 86 Pulsatile portal flow with brief pulse-synchronous flow reversal in a patient with cardiac cirrhosis. VP = portal vein.
Fig. 65 a Vena cava thrombosis (TH) in a patient with protein S and C deficiency, vena cava filter (arrows): thrombotic (TH) and poststenotic enlargement of the vena cava (VCI). b Vena cava thrombosis (VC, TH) in a patient with paraneoplastic syndrome, 32-year-old man, metastatic germ cell tumor. c Absence of flow signals (CDS) in the dilated vena cava (transverse upper abdomen section) due to a high-grade metastatic stenosis above; no thrombosis. Thrombosis 32 Venectasia Segmental dilatation of a vein may be physiological in nature or due to impaired outflow or anatomy: for example, a large vena cava lumen in adolescents, distended jugular veins, or physiological engorgement of the left renal vein when crossing the aorta.
If a white thrombus becomes superimposed on a ruptured arteriosclerotic plaque, its sonographic appearance is that of a homogeneous, hypoechoic intraluminal mass. Such plaque–thrombus complexes increase the risk of arterial occlusion (myocardial infarction), rupture, and microembolism. They also make the vessels prone to sclerosis and calcification, in which case ultrasonography will show them as heterogeneous irregular structures (Fig. 40, Fig. 46, Fig. 47). Fig. 46 Complex protruding lesion of the femoral artery (AF).